Can cerebral thrombosis cause other diseases?

Patients with cerebral thrombosis may have dizziness, headache, vomiting, numbness of one limb or limited activity, aphasia, even drowsiness, coma, hemiplegia and other symptoms.

If patients with cerebral thrombosis have serious symptoms and long-term bed care is not in place, they may be complicated with falling pneumonia, venous thrombosis of lower limbs, long-term skin pressure, poor blood supply and insufficient nutrition supply, which may lead to bedsores.

Suffering from cerebral thrombosis may also cause stress ulcer, upper gastrointestinal bleeding, central high fever and convulsion. Patients with cerebral thrombosis may cause kidney and liver damage due to long-term medication. Pay attention to control blood pressure, blood lipid and blood sugar at ordinary times, and have regular physical examination.

Cerebral thrombosis is mostly caused by cerebrovascular diseases such as atherosclerosis, and the change of blood composition is also a factor of cerebral thrombosis. Atherosclerosis is the most common lesion of cerebral artery wall and the main cause of thrombosis. Secondly, various cerebral arteritis, congenital vascular dysplasia and vascular amyloidosis are also common factors causing vascular wall lesions. Patients with cerebral thrombosis will show different clinical symptoms according to the location and area of infarction. Some patients have precursor symptoms of transient ischemic attack, such as sudden numbness and weakness of upper and lower limbs, mouth and eyes askew, slurred speech and other symptoms.

The treatment principle of cerebral thrombosis is to quickly restore the blood supply to the ischemic area, prevent the further development of thrombosis and reduce the infarct area. The treatment method is closely related to the severity of the patient's condition and whether the doctor is timely. In the treatment of acute phase, thrombolysis should be carried out within the golden time window of 4.5 hours to reduce brain metabolism, control brain edema, protect brain cells and save ischemic penumbra.