Knowledge of Cattle Raising: What's wrong with cattle being particularly thin? Is there any way?

Summary: 1, nutrition can't keep up; 2, the cause of parasites, deworming; 3. Different from the previous feeding methods, cows are in a state of emergency and should be overfeed step by step; 4, deworming and stomach strengthening, with microecological products to promote digestion and absorption, long meat will be faster.

Analysis:

1 paratuberculosis

Cattle paratuberculosis, also known as paratuberculotic enteritis, belongs to chronic intermittent diarrhea, but it lasts for a long time, and some of them show intractable diarrhea. After a long time, animals lose weight. This disease can be prevalent in ruminants, and its most obvious features are long-term, chronic and intermittent diarrhea, reduced milk production, submandibular and sarcophagus edema, but low body temperature. Autopsy showed that the intestinal mucosa of sick cattle jejunum and ileum thickened several times, forming brain-like folds, which showed necrosis, but no cheese-like substance.

2 infectious pleuropneumonia

The pathogen of this disease is mycoplasma. Sick cows with acute contagious pleuropneumonia have difficulty breathing. Animals with chronic contagious pleuropneumonia are getting thinner because of long-term intermittent and painful short cough, which leads to digestive dysfunction and poor appetite. Acute pleuropneumonia does not return to the body temperature, and chronic pleuropneumonia has only a low fever, sometimes it does not return. Due to pleural thickening and adhesion to ribs, percussion can be semi-voiced.

Pseudotuberculosis

This disease is caused by local abscess of Corynebacterium pseudotuberculosis, which spreads to the whole body in severe cases, causing multiple abscesses all over the body. Sick cows become emaciated, weak, anaemic and even die. Autopsy found that there were many abscesses in the whole body of sick cattle, which sometimes spread to the respiratory tract and caused lung cavity, body temperature rise and cough. It was similar to influenza, but the mortality rate was high, so we should pay attention to differentiation.

4 filariasis

Cattle suffering from parasitic diseases are chronic, intermittent, progressive emaciation, anemia and edema, and some animals have nervous system symptoms, especially horses. Buffalo is the most susceptible, characterized by dry skin, decreased skin temperature, and finally dry skin necrosis. In severe cases, the tail tip can be necrotic and fall off. The sick cow has swollen lower limbs and edema in the chest, which is characterized by unexplained claudication and looks like rheumatism. Sick cows have difficulty getting up or even lying down, which is similar to exhaustion.

Babesia parasitizes in red blood cells, showing ring or double bud shape, and pathogenic worms can be seen in blood films. Because this disease is transmitted by ticks, it often occurs in places where ticks are active. Emaciation is only one of the clinical manifestations of babesiosis. In addition to emaciation, the clinical features of sick cattle include the continuous increase of body temperature as high as 465,438 0.8℃, with pale mucosa, anemia, red proteinuria and edema of eyelids and limbs. Blood smear can find worms in red blood cells.

Fasciola hepatica is a chronic wasting disease. Adult parasitic on bile duct can cause cholangitis, bile duct obstruction, substantive hepatitis and cirrhosis. Clinically, sick cattle show digestive disorders, anemia, diarrhea, emaciation, subcutaneous edema of jaw and chest. The intermediate host of the disease, Verticillium spiralis, is freshwater snails. Feeding cattle with a large number of aquatic plants often leads to Fasciola hepatica. The diagnosis of this disease depends on finding Fasciola hepatica eggs in cow dung.

Trichinella spiralis, schistosomiasis, haemonchus contortus and other diseases have a chronic course, and the causes of animal wasting are unknown, which is also the first suspected parasitic infection in the diagnosis of wasting disease. In diagnosis, it is important to collect feces and detect parasite eggs, except that animals should be given proper nutritious food when they are thin. Clinical use of levamisole and other anthelmintic drugs to observe the anthelmintic effect is of great value for disease diagnosis.

5 malnutrition

Insufficient feed, being in protein for a long time, insufficient energy supply, lack of trace element cobalt in ruminants, some parasitic infections, purulent diseases in the body, and blood protozoan diseases can all accelerate the occurrence of nutritional failure. The occurrence of nutritional failure is a gradual process. The most obvious clinical symptoms of sick cattle are emaciation, hypothermia, loss of appetite, excessive emaciation, animals lying more and standing less, wobbling and lying on the ground later. Sick cows often have bedsores, thick blood and decreased serum total protein and albumin concentrations, so the disease is easy to diagnose. Friends only need to ensure that the proportion of concentrate formula is balanced and the quality of premix used is up to standard. Once the cows are found to be thin, we must carefully analyze the reasons to avoid irreversible deterioration.

6 trace element deficiency

Cobalt deficiency is a disease characterized by emaciation, which occurs in ruminants, rabbits, rats and other animals that have the habit of eating soft manure. This is because cobalt is an essential trace element for the growth and reproduction of digestive tract bacteria, and it is also an important raw material for these bacteria to synthesize cobalamin. The sick animals are emaciated, weak, pica, with fragile fur, pale mucosa and constant tears. Sheep show wet face disease. The concentration of glucose in blood decreased and the activity of alkaline phosphatase decreased. If cobalamin is added to the feed, it can quickly return to normal and establish a diagnosis. The contents of methylmalonic acid and iminomethylglutamic acid in urine increased, especially subunit glutamic acid was more sensitive.

Copper deficiency is a common disease in ruminants such as cattle and sheep, which is often caused by copper deficiency in soil environment or high molybdenum content in pasture. Animals show chronic diarrhea and emaciation for a long time, and thin feces can flow down the anus and hind legs, and be thrown to the hindquarters through the tail, polluting the coat. In addition to emaciation and diarrhea, fur fades, black turns gray, and red hair turns yellow. In particular, the hair color around the eyes is relatively light, and it looks like a pair of glasses from a distance, which is also commonly known as the glasses cow. Sheep wool is not very curved, fragile and easily broken.

Zinc deficiency, because the active ingredient of the taste substance that produces appetite is zinc, all kinds of animals lose their appetite because of zinc deficiency, and animals lose weight because of decreased appetite and insufficient food intake. Zinc-deficient animals still have the characteristics of increased dander, hair loss, chapped hoof shell and reproductive disorder.